the Technology

Ultra-Violet (UV) light is invisible to the human eye and is divided into UV-A, UV-B and UV-C. UV-C is found within 200-280 nm range. UV-C Zap Band light is focused in this spectrum which is effective against viruses and bacteria. Microorganisms effective resistance to UV light varies considerably. Moreover, the environment of the particular microorganism greatly influences the radiation dose needed for its destruction.



Does UV-C work? Yes!

There is a plethora of both scientific and anecdotal references for UV-C’s efficacy both in literature and in reports of field applications. Of the government reports, NIOSH, OSHA, CDC, GSA, EPA are the most notable. Science in the public forum comes from the University of Cincinnati, Tulsa University, University of Colorado and McGill University (Canada) to name a few. UVC is used worldwide, more in other countries per capita than in the US. Much of this broader use is for the disinfection of drinking water and treatment of sewage in wastewater treatment.

Inactivation Effectiveness:

The degree of inactivation by ultraviolet radiation is directly related to the UV dose applied. The dosage, a product of UV light intensity and exposure time, is usually measured in microjoules per square centimeter, or equivalently as microwatt seconds per square centimeter (μW·s/cm2).
Dosages for a 90% kill of most bacteria and viruses is universally regarded as greater than 2,000 μW·s/cm2.
UVC-Zap Band demonstrates a range of 2000-4000 μW·s/cm2 for a one minute pass. Coronavirus is reduced by 90% at 611 μW·s/cm2. Larger parasites such as cryptosporidium require a lower dose for inactivation. As a result, the U.S. Environmental Protection Agency has accepted UV disinfection as a method for drinking water plants to obtain cryptosporidium, giardia or virus inactivation credits. For example, for a 90% reduction of cryptosporidium, a minimum dose of 2,500 μW·s/cm2 is required based on the U.S. EPA UV Guidance Manual published in 2006.

Aftermath of ultraviolet A-B-C radiation on skin. UV-C are shielded by the atmosphere and do not reach the skin. UV-B get through the epidermis causing ROS formation (release), activation of p38 with consequent ΔNp63 detachment and leading to activation of p53 apoptotic genes. On the contrary, ΔNp63 leads a suppressive activity on p53 apoptotic function in unstimulated keratinocytes. UV-A is responsible for skin aging.